An analysis of the social and psychological explanations of schizophrenia a psychotic disorder
Biological explanations of schizophrenia
Conclusions: The results highlight the relevance of self-concepts related to social abilities, dysfunctional beliefs, and global self-worth alone and in interaction with ToM deficits for negative symptoms and thereby provide a helpful basis for advancing psychosocial interventions. More receptors lead to more firing and an over production of messages. Assessment provides an opportunity to thoroughly examine the biological, psychological and social factors that may have contributed to the onset of the illness, and also enquire about common coexisting problems such as substance misuse, anxiety, depression and physical health problems. On balance it is unlikely that any of these theories fully captures the complexity of the potential gene-environment interaction that underpins the development of psychosis and schizophrenia; see van Os et al. Post hoc plotting was used to interpret significant interactions by recasting the regression equation as the regression of the criterion on one predictor. AO3 A strength is that the research into enlarged ventricles and neurotransmitter levels have high reliability. However, despite this, considerable problems remain. Family, twin and adoption studies must be considered cautiously because they are retrospective, and diagnosis may be biased by knowledge that other family members who may have been diagnosed. ABC: Get the patients to understand what is really happening in their life: A: Antecedent - what is triggering your problem? The researchers concluded that this is more important in maintaining schizophrenia than in causing it in the first place, Brown et al Lobban reports that other family members felt they were able to cope better thanks to family therapy. Research suggests that these delusions are associated with specific biases in reasoning about and explaining social situations. Due to the pivotal role of dopamine in schizophrenia this led to a culture of heavy smoking an nicotine addiction in psychiatric hospitals of the era.
In particular, the dopamine hypothesis of psychosis has been influential and states that psychosis results from an overactivity of dopamine function in the brain, particularly in the mesolimbic pathway.
The rate of relapse was particularly high if returning to a high EE family was coupled with no medication. EE is a family communication style that involves criticism, hostility and emotional over-involvement.
For example, it could be that antipsychotic drugs cause a general neurological suppression that reduces the intensity of symptoms Moncrieff, Fowler, unpublished data, As well, patients with delusions of persecution require more time to name the color of a threatening word compared with a neutral word or a word related to depression and they tend to have a better recall of threatening words and propositions.
First, cognitive deficits which are impairments in thought processes such as perception, memory and attention. DSM should arrive at the same diagnosis for the same individual.
However, the s saw the emergence of a paradigm shift, brought about by the user movement, 1 new ideas about recovery, 13 and renewed interest in psychological treatments.
based on 83 review